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Medications

Across
Opioid analgesic; Bind to opiate receptors in the CNS. Alter the perception of and response to painful stimuli while producing generalized CNS depression
Antiplatelet; inhibits platelet aggregation by irreversibly inhibiting the binding of ATP to platelet receptors
Antiarrhythmic; increases the force of myocardial contraction
Diuretic; causes loss of sodium bicarbonate & calcium while saving potassium & hydrogen ions by antagonizing aldosterone
Antianxiety; depresses the CNS
Diuretic; (Lasix) Inhibits the reabsorption of sodium and chloride from the loop of Henle and distal renal tubule
Hormones; replacement of or supplementation to endogenous thyroid hormones
Antacid; mineral and electrolyte replacements and suspensions
Antidepressant; selectively inhibits the reuptake of serotonin in the CNS
Antihypertensive; ACE inhibitors block the conversion of angiotensin I to the vasoconstrictor angiotensin II
Antidiabetic; Lowers blood glucose by stimulating glucose uptake in skeletal muscle and fat, inhibiting hepatic glucose production
Anticoagulant; Acts as a selective, reversible site inhibitor of factor Xa, inhibiting both free and bound factor
Down
Antipyretics, Non-opioid analgesics; produce analgesia and reduce inflammation and fever by inhibiting the production of prostaglandins. Decreases platelet aggregation
Analgesic; decreases pain receptors
Antiulcer; Inhibits the action of histamine at the H2 -receptor site located primarily in gastric parietal cells, resulting in inhibition of gastric acid secretion
Analgesic adjuncts, anticonvulsants, mood stabilizers; Mechanism of action is not known. May affect transport of amino acids across and stabilize neuronal membranes
Antiulcer; inhibits the action of histamine at the H2-receptor site located primarily in gastric parietal cells, resulting in inhibition of gastric acid secretion
Antipyretics, Non-opioid analgesics; inhibits synthesis of prostaglandins that may serve as mediators of pain and fever, primarily in the CNS
Lipid lowering; Inhibits 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase, an enzyme which is responsible for catalyzing an early step in the synthesis of cholesterol
Antihypertensive; (Demadex) inhibits the reabsorption of sodium and chloride from the loop of Henle and distal renal tubule
Antianginals, antihypertensives; blocks stimulation of beta1 (myocardial)-adrenergic receptors
Lipid lowering; proposed mechanisms include ↑ activity of liver enzymes, ↓ hepatic lipogenesis, ↑ lipoprotein lipase activity and ↓ TG synthesis